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National Institutes of Health
ALTERNATING HEMIPLEGIA OF CHILDHOOD 2
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In Vivo Modelling of ATP1A3 G316S-Induced Ataxia in C. elegans Using CRISPR/Cas9-Mediated Homologous Recombination Reveals Dominant Loss of Function Defects
Ivan C. Alcantara
Corpus ID: 5543977
The NIH Undiagnosed Diseases Program admitted a male patient with unclassifiable late-onset ataxia-like symptoms. Exome…
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