• Corpus ID: 18479167

damage recovery following eccentric exercise-induced muscle Curcumin effects on inflammation and performance

@inproceedings{Davis2007damageRF,
  title={damage recovery following eccentric exercise-induced muscle Curcumin effects on inflammation and performance},
  author={J. Mark Davis and E. Angela Murphy and Martin D. Carmichael and Mark R. Zielinski and Claire M. Groschwitz and Adrienne S. Brown and J. David Gangemi and Abdul Ghaffar and Eugene P. Mayer},
  year={2007}
}
levels of biological organization, ranging from molecules to humans, including clinical investigations. It is published investigations that illuminate normal or abnormal regulation and integration of physiological mechanisms at all publishes original 
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A dietary supplement attenuates IL-6 and CRP after eccentric exercise in untrained males.

It is suggested that exercise-induced inflammation, evaluated by changes in IL-6 and CRP, was significantly reduced by the dietary supplement.

Recovery of running performance following muscle-damaging exercise: Relationship to brain IL-1β

Use of nonsteroidal anti-inflammatory drugs following exercise-induced muscle injury.

From review of the current literature, it is concluded that NSAID use for brief periods of time is beneficial for short-term recovery of muscle function and is an important laboratory tool for the study of EIMI.

Prolonged vitamin C supplementation and recovery from eccentric exercise

The results suggest that vitamin C supplementation does not affect interleukin-6 concentrations following eccentric exercise that has a low metabolic component.

Eccentric exercise-induced injury to rat skeletal muscle.

The data generally indicated that eccentric exercise causes greater injury to the muscles, and questions remain.

Changes in inflammatory mediators following eccentric exercise of the elbow flexors.

Although there was evidence of severe muscle damage after the eccentric exercise, this muscle damage was not accompanied by any large changes in plasma cytokine concentrations, and these results suggest that eccentric exercise-induced muscle damage is not associated with the significant release of cytokines into the systemic circulation.

Role of brain IL-1beta on fatigue after exercise-induced muscle damage.

The hypothesis that increased brain IL-1beta plays an important role in fatigue after muscle-damaging exercise is supported.

Indirect evidence of human skeletal muscle damage and collagen breakdown after eccentric muscle actions.

It is concluded that eccentric but not concentric actions may result in temporary muscle damage, and that collagen breakdown may also be affected by eccentric actions.

Interleukin-6 expression after repeated bouts of eccentric exercise.

Results indicate that changes in sTnI, strength and soreness were less with the second eccentric exercise bout whereas the changes in both IL-6 mRNA and protein were not effected between bouts, characteristic of the repeated bout effect.

Adaptation to lengthening contractions is independent of voluntary muscle recruitment but relies on inflammation.

It is concluded that adaptation to lengthening contractions does not depend on neural components but is likely mediated by strengthening of muscle structural/cellular elements and that inflammation is important for this process.