and RA affects gastric mucosal superoxide dismutases Treatment of Helicobacter pylori infection favourably

Abstract

Background and aims—Excessive production of reactive oxygen metabolites (ROMs) by phagocytic cells is thought to contribute to the mucosal pathology of Helicobacter pylori infection. Previously, H pylori infection was shown to have a differential effect on some gastric mucosal scavenger enzymes of ROMs – namely, mitochondrial and cytoplasmic superoxide… (More)

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