What is myocarditis?

@article{Olsen2005WhatIM,
  title={What is myocarditis?},
  author={Eckhardt G. J. Olsen},
  journal={Heart and Vessels},
  year={2005},
  volume={1},
  pages={1-3}
}
  • E. Olsen
  • Published 1 March 1985
  • Medicine, Biology
  • Heart and Vessels
SummaryA brief historical review of the concepts that have existed in myocarditis in the nineteenth and twentieth centuries is presented. The establishment of this entity is linked with the milestones of discovery in medicine, especially the development of the stethoscope, the microscope, and the science of bacteriology. Yet, despite these advances, chronic myocarditis was still a common diagnosis up to about the third decade of the twentieth century and included conditions such as hypertension… 

Histopathologic aspects of viral myocarditis and its diagnostic criteria

  • E. Olsen
  • Medicine, Biology
    Heart and Vessels
  • 2005
It has been emphasized that clear concepts and a uniform approach are essential in diagnosing myocarditis, not only to achieve precise diagnosis, accurately monitoring the response to therapy, but also in helping to establish the pathogenetic pathways for certain forms of cardiomyopathy.

Role of CD1d in Coxsackievirus B3-Induced Myocarditis 1

It is demonstrated thatCD1d expression is essential for pathogenicity of CVB3-induced myocarditis, that CD1D expression is increased early after infection in vivo in CD1d+ mice infected with the myocarditic but not with the nonmyocarditicCVB3 variant, and that Vγ4+ T cells, which are known to promoteMyocarditis susceptibility, appear to recognize CD1 d expressed by CVB 3-infected myocytes.

Myocarditisof CD1d in Coxsackievirus B3-Induced

This article cites 42 articles, 29 of which you can access for free at: Subscriptions http://jimmunol.org/content/170/6/3147.full#ref-list-1.

The effects of perindopril on QT duration and dispersion in patients with coronary slow flow

Coronary slow may be associated with prolonged QT interval and increased QT dispersion and impaired diastolic filling and perindopril may be helpful in restoration of these findings.

Coxsackievirus B3 replication and pathogenesis.

Viruses such as coxsackievirus B3 (CVB3) are entirely host cell-dependent parasites. Indeed, they must cleverly exploit various compartments of host cells to complete their life cycle, and

Host protein manipulation as a mechanism in viral cardiomyopathy

This paper presents a meta-analysis of the literature on coxsackievirus-encoded proteins and its role in the pathogenesis and treatment of viral myocarditis, which is a tri-phasic disease with in vitro and In vivo CVB3 infection.

References

SHOWING 1-10 OF 18 REFERENCES

Myocarditis--a case of mistaken identity?

  • E. Olsen
  • Medicine
    British heart journal
  • 1983
Often, on occasions such as this, one is informed that a person of note, hitherto believed to have succumbed to a certain disease, has in fact died as a result of a totally different disease, which is of course the subject of this lecture.

Coxsackie B myopericarditis in adults.

  • W. Smith
  • Medicine
    American heart journal
  • 1970

HYPERTENSION HEART: THE MOST COMMON FORM OF SO-CALLED CHRONIC MYOCARDITIS

If the authors are to prevent heart disease in the future, or even increase the expectancy of persons having it, they must increase the knowledge of the factors concerned in its production.

Cardiomyopathies: Pathology and Infectious Immune Mechanisms

Cardiomyopathies are now defined as “heart muscle diseases of unknown cause” and classified into three major types: dilated, hypertrophic and restrictive.

CLINICAL FEATURES OF SUDDEN OBSTRUCTION OF THE CORONARY ARTERIES

In Germany Cohnheim supported the views of Hyrtl and Henle as to lack of considerable anastomosis, and as late as 1881 lent the influence of his name to the doctrine that the coronary arteries were end-arteries.

Endomyocardial biopsy.

  • R. Shabetai
  • Medicine
    Giornale italiano di cardiologia
  • 1989

Panel Discussion

  • D. Blei
  • Computer Science
    SNA@ICML
  • 2006

Non - rheumatic myocarditis and miscellaneous cardiopathies

  • 1968

Endomyocardial fibrosis and eosinophilic heart disease

  • 1979