What's in a name?

@article{Forest1995WhatsIA,
  title={What's in a name?},
  author={White Spruce Forest},
  journal={Environmental Health Perspectives},
  year={1995},
  volume={103},
  pages={1080 - 1080}
}
  • W. Forest
  • Published 1 December 1995
  • Chemistry
  • Environmental Health Perspectives
Kathryn Rosica of the Chemical Manufacturers Association raised an interesting point in her letter (EHP vol. 102, p. 1006). Neither the term "glycol ethers" nor the term "ethylene glycol ethers" strictly identifies a class of chemicals whose members all share a common distinctive toxicological profile. As Rosica correctly noted, "Higher molecular weight ethylene glycol monoethers that have been tested have not been associated with significant adverse developmental and reproductive effects." For… 

References

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  • K. Uemura
  • Medicine
    Nihon Sanka Fujinka Gakkai zasshi
  • 1980
Ethylene glycol dimethyl ether was administered to pregnant mice on the 7th, 8th, 9th, and 10th days of pregnancy for examination of its effect on feti, with special reference to the presence or absence of teratogenicity.

Experimental studies on toxicity of ethylene glycol alkyl ethers in Japan.

Past studies on the toxicological effects of ethylene glycol alkyl ethers as well as the recent data on these chemicals in Japan are reviewed and there is no convincing evidence that propylene gly col monomethyl ether, diethylene Glycol monometHyl ether or diethane glycol dimethyl ether causes testicular atrophy in mice.

Developmental toxicity of nine selected compounds following prenatal exposure in the mouse: naphthalene, p-nitrophenol, sodium selenite, dimethyl phthalate, ethylenethiourea, and four glycol ether derivatives.

Results provide guidelines for the subsequent toxicity testing of these chemicals and indicate that SS, NAP, and PNP were more toxic than the polyglycol ethers, ETU, and DMP.

Results of testing fifteen glycol ethers in a short-term in vivo reproductive toxicity assay.

Fifteen glycol ethers were investigated for their potential to cause adverse reproductive toxic effects using an in vivo mouse screening bioassay to identify, for each chemical, the maternal LD10 as the target dose.