Vitamin K: the missing link to prostate health.

  title={Vitamin K: the missing link to prostate health.},
  author={Michael S. Donaldson},
  journal={Medical hypotheses},
  volume={84 3},

Vitamin K2: Implications for Cardiovascular Health in the Context of Plant-Based Diets, with Applications for Prostate Health

The process of varicose vein formation includes a role for vitamin K, implicating a lack of vitamin K in the development of varicoceles, which leads to benign prostate hyperplasia and it is likely that much good will be accomplished using vitamin K2 in interventions.

Nutritional supplementation of the pharmacotherapy of prostate diseases

The evidence base of the vitamin D prostatotropic effects has been accumulated, which allows us to consider its deficiency replacement as an effective nutritional supplement in prostate diseases.

Prospective evaluation of the association between varicocele and benign prostatic hyperplasia in men over 40 years of age

There was a statistically significant difference between the groups in terms of mean age, post‐void residual, International Prostate Symptom Score and PSA, but no differences were found in the percentage of patients with varicocele when comparing men with prostates smaller than 40 ml and greater than or equal to 40 ml.



Reversal of benign prostate hyperplasia by selective occlusion of impaired venous drainage in the male reproductive system: novel mechanism, new treatment

A novel and tested pathophysiological mechanism for the evolution of BPH is proposed and a tested and effective treatment is suggested that restores normal pressure in the venous drainage in the male reproductive system.

5α-Reductase Inhibition in the Treatment of LUTS and BPH: Update and Importance of Dual Inhibition of Types 1 and 2

The articles in this supplement issue of Reviews in Urology illuminate the role of one pivotal metabolic step in the etiology and pathophysiology of benign prostatic hyperplasia (BPH) and associated

Components of the metabolic syndrome—risk factors for the development of benign prostatic hyperplasia

It is concluded that NIDDM, treated hypertension, obesity, low HDL-cholesterol levels and high insulin levels constitute risk factors for the development of BPH and that BPH patients may share the same metabolic abnormalities of a defective insulin-mediated glucose uptake and secondary hyperinsulinemia as patients with the metabolic syndrome.

Relationship between serum sex hormones levels and degree of benign prostate hyperplasia in Chinese aging men.

The effects of endocrine changes on measures of BPH in aging men require further investigation in longitudinal and multicenter studies that include patients with all severities of B PH.

Benign prostatic hyperplasia: an overview.

The hormonal hypothesis has many arguments in its favor for treatment of BPH, and tissue ablative surgical treatment options are superior to both minimally invasive and medical therapy.

Dietary intake of menaquinone is associated with a reduced risk of coronary heart disease: the Rotterdam Study.

It is suggested that an adequate intake of menaquinone could be important for CHD prevention and not related to any of the outcomes.

Is varicocele prevalence increasing with age?

It is found that the prevalence of varicocele increases with age with a rise of about 10% for each decade of life with the incidence reaching 75% in the eight decade oflife.

Chronic Venous Insufficiency: Worldwide Results of the RELIEF Study

The RELIEF study provided important information about the epidemiology and clinical manifestations of CVI, with the observation that venous reflux was found to be absent in 57% of patients diagnosed as suffering from CVI belonging to CEAP classes C0 to C4.

Mechanisms of varicose vein formation: valve dysfunction and wall dilation

Abstract Varicose veins are a common venous disease of the lower extremity. Although the mechanisms and determinants in the development of varicosities are not clearly defined, recent clinical

Role of saphenous vein wall in the pathogenesis of primary varicose veins.

The theory of primary weakness of the vein wall as a cause of varicosity is supported, due to intimal changes, disturbance in the connective tissue components and smooth muscle cells.