Viral load, E2 gene disruption status, and lineage of human papillomavirus type 16 infection in cervical neoplasia.

@article{Cheung2006ViralLE,
  title={Viral load, E2 gene disruption status, and lineage of human papillomavirus type 16 infection in cervical neoplasia.},
  author={Jo L.K. Cheung and Keith Wing-kit Lo and Tak Hong Cheung and J. W. Tang and Paul Chan},
  journal={The Journal of infectious diseases},
  year={2006},
  volume={194 12},
  pages={
          1706-12
        }
}
The clinical utility of human papillomavirus (HPV) load and integration status remains unclear. We applied refined methods to delineate the viral load, integration status, and lineage of 104 women with HPV-16 monotype infection, including 19 with normal cervices, 9 with histologically proven cervical intraepithelial neoplasia (CIN) 1, 24 with CIN 2, 27 with CIN 3, and 25 with squamous cell carcinoma (SCC). Higher crude viral load, as determined by real-time polymerase chain reaction (PCR… CONTINUE READING
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