• Corpus ID: 115469838

Ventricular torsional relation to ventricular fiber arrangement

@article{Ranjbar2014VentricularTR,
  title={Ventricular torsional relation to ventricular fiber arrangement},
  author={Saeed Ranjbar and Tohid Emami Meybodi and Mahmood Emami Meybodi},
  journal={arXiv: Medical Physics},
  year={2014}
}
Left ventricular torsion from helically oriented myofibers is a key parameter of cardiac performance. Physicians observing heart motion on echocardiograms, during cardiac catheterization, or in the operating room, are impressed by the twisting or rotary motion of the left ventricle during systole. Conceptually, the heart has been treated as a pressure chamber. The rotary or torsional deformation has been poorly understood by basic scientists and has lacked clinical relevance. The aim of this… 
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References

SHOWING 1-10 OF 41 REFERENCES

Spatial orientation of the ventricular muscle band: physiologic contribution and surgical implications.

It is hypothesized that the ventricular myocardium, both right (RV) and left (LV), exists as a continuous muscle band and this unique anatomy and spatial configuration of the myocardial muscle determine the way theventricular ejection and filling take place.

Myocardial fiber architecture of the human heart ventricles

The ventricular fiber architecture has been studied anatomically and histologically in 25 human hearts and the results have attempted to correlate the ventricular architecture with the valvular dynamics.

Fiber Orientation in the Canine Left Ventricle during Diastole and Systole

The wall has a well-ordered distribution of fiber angles varying from about 60° (from the circumferential direction) at the inner surface to about –60° on the outer surface, and the greatest change in angle occurs at the two surfaces (endocardial and epicardial).

Relating myocardial laminar architecture to shear strain and muscle fiber orientation.

sheets coincide with planes of maximum systolic shear subject to the constraint that the muscle fiber axis is contained in this plane, suggesting that two populations of sheet orientation may exist.

The helical ventricular myocardial band: global, three-dimensional, functional architecture of the ventricular myocardium.

  • M. KocicaA. Corno F. Torrent-Guasp
  • Medicine
    European journal of cardio-thoracic surgery : official journal of the European Association for Cardio-thoracic Surgery
  • 2006

Regional ventricular wall thickening reflects changes in cardiac fiber and sheet structure during contraction: quantification with diffusion tensor MRI.

It is shown for the first time that geometric changes in both sheet and fiber orientation provide a substantial mechanism for radial wall thickening independent of active components due to myofiber shortening.

The helical ventricular myocardial band of Torrent-Guasp: potential implications in congenital heart defects.

  • A. CornoM. KocicaF. Torrent-Guasp
  • Medicine
    European journal of cardio-thoracic surgery : official journal of the European Association for Cardio-thoracic Surgery
  • 2006

Characterization of the normal cardiac myofiber field in goat measured with MR-diffusion tensor imaging.

The transmural course of the helix angle corresponded to data reported in literature and appeared to be <0.07 mm(-1) throughout the myocardial walls except for the fusion sites between the left and right ventricles and the insertion sites of the papillary muscles.

Transverse shear along myocardial cleavage planes provides a mechanism for normal systolic wall thickening.

The data indicate that rearrangement of myocytes by slippage along myocardial cleavage planes is in the correct direction and of sufficient magnitude in the subendocardium (inner third) to account for a substantial proportion of systolic wall thickening.

Cellular basis for volume related wall thickness changes in the rat left ventricle.