Ventricular performance after onset of renal hypertension.

@article{Ferrario1976VentricularPA,
  title={Ventricular performance after onset of renal hypertension.},
  author={Carlos M. Ferrario and Anthony Kosoglov and R Bajbus and George R. Nadzam},
  journal={Clinical science and molecular medicine. Supplement},
  year={1976},
  volume={3},
  pages={
          141s-143s
        }
}
1. In renal hypertensive dogs, the slope of the relation between either stroke volume or cardiac output and end-diastolic pressure is shifted to the right of the normal curves. 2. The maximum to which stroke volume or cardiac output can be raised during loading is significantly less. These changes in cardiac performance appear to be unrelated to the severity of hypertension. 
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Cardiac pumping ability in rats with experimental renal and genetic hypertension.
Basal inotropic state in rats with renal hypertension: influence of coronary flow and perfusion pressure.
Cardiac performance in renal hypertensive animals has been variously reported as normal, increased, or decreased. Because of the many factors that can alter cardiac function in vivo ventricular…
Cardiac Pumping Ability Following Reversal of Hypertrophy and Hypertension in Spontaneously Hypertensive Rats
TLDR
The data indicate that treatment with methyldopa is associated with improved ventricular function in part related to the reduction in arterial pressure.
Hemodynamic consequences of left ventricular hypertrophy in spontaneously hypertensive rats.
Cardiac and vascular structural adaptation in experimental hypertension.
TLDR
At low aortic pressures and hence limited coronary perfusion, LV performance is attenuated in SHR and renal hypertensive rats, most likely due to the vascular structural changes within the coronary vascular bed.
Echocardiographic evidence for increased cardiac performance during early renal hypertension in dogs.
TLDR
Cardiac function early (less than 4 weeks) in the course of experimental renal hypertension was investigated in 11 male mongrel dogs, and it was suggested that increased myocardial contractility accounted for the findings.
Functional, morphological and metabolic characteristics of isolated hearts from normotensive and spontaneously hypertensive rats before, during and after renal hypertension.
TLDR
It is concluded that LVH generally enhances cardiac performance, but that concomitant renal hypertension exerts a cardio-depressive influence which can neither be ascribed to an increased fibrous tissue content nor to a reduced energy charge potential.
Myocardial Biochemical, Contractile, and Electrical Performance after Imposition of Hypertension in Young and Old Rats
TLDR
The Stress of hypertension, which was tolerated by the 10-and 20-month-old animals with lesser relative hypertrophy and lesser changes in measured parameters, may represent a differential adaptation to the stress of hypertension.
Key Word: left ventricular function - myocardial contractility - intrinsic heart rate - plasma catecholamines - M-mode echocardiography .
TLDR
Cardiac function early in the course of experimental renal hypertension was investigated in 11 male mongrel dogs to produce two-kidney, one-wrapped hypertension, suggesting that inereaged cardiac contractility accounted for the findings.
Current views concerning cardiac output in the genesis of experimental hypertension.
TLDR
This review is directed to the analysis of hemodynamic factors in experimental hypertensive disease and shows that arterial hypertension of various origins and duration is accompanied by various hemodynamic abnormalities; their characteristics are increased peripheral resistance with normal to low cardiac output and a transitional pattern of an early rise in cardiac output with a latter progressive increase in peripheral resistance.
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References

SHOWING 1-5 OF 5 REFERENCES
Cardiac Performance in Rats with Renal Hypertension
TLDR
The data indicate that chronic renal hypertension is accompanied by a depression of cardiac reserve which is not revealed by measurements of cardiac output and left ventricular end-diastolic pressure at rest, and this impairment in cardiac function might be related to either diminished cardiac contractility or reducedleft ventricular compliance.
Increased Cardiac Output as a Contributory Factor in Experimental Renal Hypertension in Dogs
TLDR
Increased cardiac output probably has a contributory role in the development of renal hypertension, and absence of change in plasma volume and increase in mean circulatory pressure measured in a separate series of 7 dogs suggest that the initial increase in cardiac output preceding rise in pressure was due to enhanced venous return consequent to constriction of capacitance vessels.
Quantitative Description of Ventricular Output Curves in Conscious Dogs
TLDR
Ventricular output curves obtained from normal dogs, chronic cardiac-denervated dogs, and dogs with beta-adrenergic blockade were analyzed in terms of a compartmental mathematical model and a quantitation of ventricular function from ventricular output curves can be better approached.
Circulatory Control in Hypertension
TLDR
The analysis predicts that changes in renal resistance or in other aspects of renal function that affect the ability of the kidneys to eliminate water and salt will have profound effects on long-term regulation of arterial pressure.
On the differences and similarities between the benign and malignant phases of renal hypertension
  • 1975