Ventral pallidum mediates amygdala-evoked deficits in prepulse inhibition.

@article{Forcelli2012VentralPM,
  title={Ventral pallidum mediates amygdala-evoked deficits in prepulse inhibition.},
  author={Patrick A Forcelli and Elizabeth A West and Alice T Murnen and Ludise M{\'a}lkov{\'a}},
  journal={Behavioral neuroscience},
  year={2012},
  volume={126 2},
  pages={290-300}
}
Prepulse inhibition (PPI) is an operational measure of sensorimotor gating. It is defined as a reduction in magnitude of a startle response when a startling stimulus is preceded by a weaker "prepulse." PPI has been found to be altered in patients with schizophrenia, autism spectrum disorders, and other neuropsychiatric illnesses. As such, the neural substrates regulating PPI are of particular interest. Previous studies using lesions, selective blockade of N-methyl-d-aspartate (NMDA) receptors… CONTINUE READING
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