Vascular effects of delta 9-tetrahydrocannabinol (THC), anandamide and N-arachidonoyldopamine (NADA) in the rat isolated aorta.

@article{OSullivan2005VascularEO,
  title={Vascular effects of delta 9-tetrahydrocannabinol (THC), anandamide and N-arachidonoyldopamine (NADA) in the rat isolated aorta.},
  author={Saoirse Elizabeth O'Sullivan and David A. Kendall and Michael D Randall},
  journal={European journal of pharmacology},
  year={2005},
  volume={507 1-3},
  pages={
          211-21
        }
}

Time-Dependent Vascular Effects of Endocannabinoids Mediated by Peroxisome Proliferator-Activated Receptor Gamma (PPARγ)

Data indicate that endocannabinoids cause time-dependent, PPARγ-mediated vasorelaxation, which may represent a novel mechanism by which endoc cannabinoidoids are involved in vascular regulation.

Δ9-tetrahydrocannabinol treatment improved endothelium-dependent relaxation on streptozotocin/nicotinamide-induced diabetic rat aorta.

Results suggested that THC improved endothelium-dependent relaxation in STZ/NIC induced diabetic rat aorta and that these effects were mediated at least in part, by control of hyperglycemia and enhanced endothelial nitric oxide bioavailability.

Effects of hypertension on vasorelaxation to endocannabinoids in vitro.

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