Varicella-zoster virus T cell tropism and the pathogenesis of skin infection.

@article{Arvin2010VaricellazosterVT,
  title={Varicella-zoster virus T cell tropism and the pathogenesis of skin infection.},
  author={Ann Margaret Arvin and Jennifer F. Moffat and Marvin H Sommer and Stefan L. Oliver and Xibing Che and Susan E. Vleck and Leigh Zerboni and Chia-Chi Ku},
  journal={Current topics in microbiology and immunology},
  year={2010},
  volume={342},
  pages={189-209}
}
Varicella-zoster virus (VZV) is a medically important human alphaherpesvirus that causes varicella and zoster. VZV initiates primary infection by inoculation of the respiratory mucosa. In the course of primary infection, VZV establishes a life-long persistence in sensory ganglia; VZV reactivation from latency may result in zoster in healthy and immunocompromised patients. The VZV genome has at least 70 known or predicted open reading frames (ORFs), but understanding how these gene products… CONTINUE READING

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