Uterine estrogen receptors are increased by RU486 in late pregnant rhesus macaques but not after spontaneous labor.

@article{Haluska1990UterineER,
  title={Uterine estrogen receptors are increased by RU486 in late pregnant rhesus macaques but not after spontaneous labor.},
  author={George J Haluska and N B West and Miles J. Novy and Robert M. Brenner},
  journal={The Journal of clinical endocrinology and metabolism},
  year={1990},
  volume={70 1},
  pages={181-6}
}
Progesterone withdrawal as a mechanism for parturition in primates is controversial. The progesterone antagonist RU486, given in late pregnancy to rhesus monkeys at a dose of 47 mmol/kg.day (20 mg/kg.day), causes an increase in uterine activity, but not the expected increase in amniotic fluid prostaglandins or cervical dilatation. We, therefore, studied the effect of RU486 on estrogen receptor (ER) localization and concentration in reproductive tract tissues in rhesus monkeys during late… CONTINUE READING

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