Update on the pathophysiology and classification of von Willebrand disease: a report of the Subcommittee on von Willebrand Factor.

@article{Sadler2006UpdateOT,
  title={Update on the pathophysiology and classification of von Willebrand disease: a report of the Subcommittee on von Willebrand Factor.},
  author={J Evan Sadler and Ulrich Budde and Jeroen C J Eikenboom and Emmanuel J Favaloro and Frank G. H. Hill and Lars Holmberg and Jo̸rgen Ingerslev and ChristineA. Lee and David Lillicrap and Pier Mannuccio Mannucci and Claudine Mazurier and Dominique Meyer and William L. Nichols and Mizuki Nishino and Ian Peake and Francesco Rodeghiero and Reinhard Schneppenheim and Zaverio M. Ruggeri and Alok Prasad Srivastava and Robert R. Montgomery and Augusto B Federici},
  journal={Journal of thrombosis and haemostasis : JTH},
  year={2006},
  volume={4 10},
  pages={
          2103-14
        }
}
von Willebrand disease (VWD) is a bleeding disorder caused by inherited defects in the concentration, structure, or function of von Willebrand factor (VWF). VWD is classified into three primary categories. Type 1 includes partial quantitative deficiency, type 2 includes qualitative defects, and type 3 includes virtually complete deficiency of VWF. VWD type 2 is divided into four secondary categories. Type 2A includes variants with decreased platelet adhesion caused by selective deficiency of… CONTINUE READING
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