Unrecognized peripartum cardiomyopathy in Haitian women

@article{Fett2005UnrecognizedPC,
  title={Unrecognized peripartum cardiomyopathy in Haitian women},
  author={James D. Fett and Leonard G. Christie and Robert D. Carraway and Aftab A. Ansari and J. Bruce Sundstrom and J. G. Murphy},
  journal={International Journal of Gynecology \& Obstetrics},
  year={2005},
  volume={90}
}
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TLDR
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5 Peripartum Cardiomyopathy : A Systematic Review
TLDR
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TLDR
Because of its rare incidence, geographical differences, and heterogeneous presentation, PPCM continues to be incompletely characterized and understood, so future epidemiological trials and national registries are needed to learn more about the disease.
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RETROSPECTIVE ANALYSIS OF INCIDENCE AND OUTCOME OF PERIPARTUM CARDIOMYOPATHY PATIENTS IN EMERGENCY OBSTERTIC CASES IN RURAL TERTIARY CARE HOSPITAL
TLDR
The aim of the study was to determine the incidence and outcome of peripartum cardiomyopathy patients in emergency obstetric cases over one and half year period in the authors' college hospital.
Peripartum women with dyspnea in the emergency department
TLDR
Patients with PPCM had a higher heart rate, and the white blood cell, high-sensitivity C-reactive protein, and B-type natriuretic peptide levels were markedly elevated, whereas PaO2 and left ventricular ejection fraction (LVEF) were lower.
Peripartum cardiomyopathy: An analysis of clinical profiles and outcomes from a tertiary care centre in southern India
TLDR
Peripartum cardiomyopathy with poor left ventricular ejection fraction and shock is associated with adverse maternal outcomes, while non-severe maternal anaemia predisposes to adverse fetal outcomes.
Peripartum Cardiomyopathy: a Review for the Clinician
TLDR
An increase in oxidative stress and anti-angiogenic activity play key roles in the pathophysiology of peripartum cardiomyopathy andTherapies that target this dysregulation may have a future role in treatment.
Pathophysiology and epidemiology of peripartum cardiomyopathy
TLDR
The latest understanding of the epidemiology, pathophysiology, and novel treatment strategies for patients with peripartum cardiomyopathy are described.
Practical management of peripartum cardiomyopathy
TLDR
A multidisciplinary team approach is essential for acute HF, antepartum, labor and postpartum care, and recovery from left ventricular dysfunction is critical for prognosis.
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References

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Emerging insights into peripartum cardiomyopathy.
TLDR
Although the cause of PPCM and reasons for increased incidence in the HAS district of Haiti remain unknown, initial data present emerging insights and avenues to pursue in subsequent studies.
Peripartum cardiomyopathy in the Hospital Albert Schweitzer District of Haiti.
TLDR
Peripartum cardiomyopathy appears to be relatively common in the Hospital Albert Schweitzer District of Haiti and a core group of patients is identified for ongoing epidemiologic and immunohematologic investigation of risk factors and potential etiologic factors.
[Two cases of peripartum cardiomyopathy].
TLDR
These two cases suggest the importance of evaluation of endomyocardial biopsy to determine the degree of interstitial fibrosis that may reflect the prognosis for patients with peripartum cardiomyopathy confirmed by measurements of ejection fraction.
Autoimmune mechanisms as the basis for human peripartum cardiomyopathy
TLDR
Findings for the first time suggest that such abnormalities may in concert lead to the initiation and perpetuation of an autoimmune process, which leads to cardiac failure and disease.
Peripartum cardiomyopathy: National Heart, Lung, and Blood Institute and Office of Rare Diseases (National Institutes of Health) workshop recommendations and review.
TLDR
Peripartum cardiomyopathy is a rare lethal disease about which little is known and diagnosis is confined to a narrow period and requires echocardiographic evidence of left ventricular systolic dysfunction.
Inhibition of Progenitor Dendritic Cell Maturation by Plasma from Patients with Peripartum Cardiomyopathy: Role in Pregnancy-associated Heart Disease
TLDR
In vitro maturation of normal human cord blood derived progenitor dendritic cells (CBDCs) was significantly reduced, marking the first findings linking inhibition of DC maturation to the dysregulation of normal physiologic cardiac tissue remodeling during pregnancy and the pathogenesis of PPCM.
Direct evidence for a β1-adrenergic receptor–directed autoimmune attack as a cause of idiopathic dilated cardiomyopathy
Today, dilated cardiomyopathy (DCM) represents the main cause of severe heart failure and disability in younger adults and thus is a challenge for public health. About 30% of DCM cases are genetic in
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