Twenty-nine years after carbon monoxide intoxication

@article{Roohi2001TwentynineYA,
  title={Twenty-nine years after carbon monoxide intoxication},
  author={Fereydoon Fred Roohi and Roger W. Kula and Nirav B. Mehta},
  journal={Clinical Neurology and Neurosurgery},
  year={2001},
  volume={103},
  pages={92-95}
}
Carbon monoxide (CO) is a worldwide environmental toxin and a leading cause of deliberate or accidental poisoning. There is an extensive literature devoted to the clinical features and treatment of those victims who have survived acute CO poisoning for a short length of time. The long-term sequelae of non-fatal poisoning have received scanty references, and the prospects for the long-lasting survivors of acute CO intoxication are less clear. Literature review uncovered reports of only three… 
The Diversity of Carbon Monoxide Intoxication: Medical Courses Can Differ Extremely—A Case Report
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It is shown that, in spite of almost equal serum levels of carboxyhemoglobin (COHb), the individual symptoms can vary extremely and a detailed medical history, physical examination, supporting diagnostic measures, and the continuous monitoring of vital parameters in a specialized clinic are essential.
Carbon Monoxide Poisoning and Cognitive Impairment
TLDR
Cognitive impairments following CO poisoning, mechanism of injury including the role of hypoxia, and treatment for cognitive impairments are described.
Myocardial injury and long-term mortality following moderate to severe carbon monoxide poisoning.
TLDR
Myocardial injury occurs frequently in patients hospitalized for moderate to severe CO poisoning and is a significant predictor of mortality.
Carbon monoxide intoxication: What we know.
TLDR
The epidemiology, symptoms, diagnosis and the therapy of this multidisciplinary challenge involving multiple organ systems without a typical clinical presentation is reviewed.
Magnetic resonance imaging findings in chronic carbon monoxide intoxication
TLDR
Patients with severe CO intoxication may develop persistent cerebral changes independently of their neuropsychiatric findings in the chronic stage, even if asymptomatic, according to the MRI findings of chronic stage CO intoxication.
Hemorheological changes in cerebral circulation of rabbits with acute carbon monoxide poisoning.
TLDR
The results suggest that the changes in hemorheology participate in the development of acute CO poisoning, which may play a role in delayed encephalopathy after acuteCO poisoning.
Glasgow Coma Scale and laboratory markers are superior to COHb in predicting CO intoxication severity.
TLDR
It is postulate that clinical parameters, such as the Glasgow Coma Scale and the laboratory markers CRP and leucocyte count are adequate supportive tools for evaluating the severity of CO-related illness, and that the measurement of COHb alone is insufficient for this purpose.
COHgb levels predict the long-term development of acute myocardial infarction in CO poisoning.
TLDR
In patients presenting to the ED with CO poisoning, COHb levels can be helpful for risk stratification in the long-term development of AMI.
Transient tic disorder following carbon monoxide poisoning.
We report a 12-year-old male patient who developed transient motor and vocal tics twelve days after carbon monoxide (CO) poisoning. Cranial magnetic resonance image (MRI) of the patient showed
Seizures and Unilateral Cystic Lesion of the Basal Ganglia: an Unusual Clinical and Radiological Manifestation of Chronic Non-fatal Carbon Monoxide (CO) Poisoning
TLDR
A case of chronic non-fatal CO poisoning presenting with epilepsy in whom magnetic resonance imaging of the brain showed a unilateral cystic lesion in the basal ganglia with bilateral dilatation of temporal horns suggestive of hippocampal atrophy is presented.
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