Translamina Cribrosa Pressure Difference as Potential Element in the Pathogenesis of Glaucomatous Optic Neuropathy.

@article{Jonas2016TranslaminaCP,
  title={Translamina Cribrosa Pressure Difference as Potential Element in the Pathogenesis of Glaucomatous Optic Neuropathy.},
  author={Jost Bruno Jonas and Ningli Wang and Diya Yang},
  journal={Asia-Pacific journal of ophthalmology},
  year={2016},
  volume={5 1},
  pages={5-10}
}
The main proven risk factor for glaucomatous optic neuropathy (GON) is an intraocular pressure (IOP) higher than the pressure sensibility of the optic nerve head allows. Fulfilling Koch postulates, numerous studies have shown that the presence of high IOP leads to GON, that lowering IOP stops the progression of GON, and that a re-increase in IOP again causes the progression of GON. There are, however, many patients with glaucoma who have statistically normal or low IOP, and despite low IOP… CONTINUE READING
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