Toward understanding renal Fanconi syndrome: step by step advances through experimental models.

@article{Sirac2011TowardUR,
  title={Toward understanding renal Fanconi syndrome: step by step advances through experimental models.},
  author={Christophe Sirac and Frank Bridoux and Marie Essig and Olivier Devuyst and Guy Touchard and Michel Cogn{\'e}},
  journal={Contributions to nephrology},
  year={2011},
  volume={169},
  pages={247-61}
}
Renal Fanconi syndrome (FS) is a generalized dysfunction of proximal tubular epithelial cells leading to the urinary leak of essential metabolites like phosphate, uric acid, glucose, amino acids and low molecular weight proteins. From inherited forms involving mutations on apparently unrelated genes to acquired forms induced by drugs, heavy metals or monoclonal immunoglobulin (Ig) light chains (LC), heterogeneous causalities of FS have complicated the understanding of this pathology for a long… CONTINUE READING
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