Tomographie par émission de positons dans la migraine

@article{Graud2005TomographieP,
  title={Tomographie par {\'e}mission de positons dans la migraine},
  author={Gilles G{\'e}raud and Marie Denuelle and Nelly Fabre and Pierre Payoux and François Chollet},
  journal={Revue Neurologique},
  year={2005},
  volume={161},
  pages={666-670}
}
Neuroimaging in headache.
Posterior Cerebral Hypoperfusion in Migraine Without Aura
TLDR
Significant relative bilateral posterior cortical hypoperfusion was found and persisted after headache relief following sumatriptan injection within 4 h after the attack onset, suggesting a common pathogenesis in migraine with and without aura.
Analysis of the Pathomechanism and Treatment of Migraines Related to the Role of the Neuropeptide CGRP
TLDR
It will become apparent as to what research should be conducted to further the field of study on migraines based on what related topics have not been currently explored in depth in other studies through a summary of the compilation of various studies conducted.
Structural and Functional Brain Changes in Migraine
TLDR
A better understanding of alterations in the brains of patients with migraine could help not only in the diagnosis but may potentially lead to the optimization of a targeted anti-migraine therapy.
Structural and Functional Neuroimaging in Migraine: Insights From 3 Decades of Research
TLDR
Three main categories of structural and functional brain changes are described in this review, corresponding to the migrainous aura, ictal headache, and interictal states, and greatly advance the understanding of multiple pathophysiologic underpinnings of migraine.
Role of neuropeptides in migraine: where do they stand in the latest expert recommendations in migraine treatment?
TLDR
Experimental and clinical investigations focused on neuropeptide involvement in migraine are reviewed, including CGRP (calcitonin gene‐related peptide), which is associated with headache and cluster headache.
Migraine: an endemic disease inside the blood–brain barrier
TLDR
The future introduction of CGRP antagonists in clinical practice could represent significant progress for acute migraine therapy, both with respect to its pathogenesis and how to effectively treat its symptoms.
Preventive Treatment with Lomerizine Increases Cerebral Blood Flows during the Interictal Phase of Migraine.
  • K. Ikeda, J. Aoyagi, Y. Iwasaki
  • Medicine, Psychology
    Journal of stroke and cerebrovascular diseases : the official journal of National Stroke Association
  • 2018
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References

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Brief report: bilateral spreading cerebral hypoperfusion during spontaneous migraine headache.
TLDR
A series of patients who had migraine headaches during serial cerebral blood-flow measurement by the intracarotid xenon-133 technique observed a pattern of localized decreases in flow that appeared to spread contiguously along the cerebral cortex.
A PET study exploring the laterality of brainstem activation in migraine using glyceryl trinitrate.
TLDR
Results suggest that lateralization of pain in migraine is due to lateralized brain dysfunction, and the activation persisted after pain was controlled by sumatriptan.
Common Migraine Attacks Preceded by Focal Hyperemia and Parietal Oligemia in the rCBF Pattern
  • H. Gelmers
  • Medicine
    Cephalalgia : an international journal of headache
  • 1982
TLDR
Results indicate that the initiating cerebral hemodynamics in common migraine are, at least in particular patients, the same as in patients with classic migraine.
Brain stem activation in spontaneous human migraine attacks
TLDR
Findings support the idea that the pathogenesis of migraine is related to an imbalance in activity between brain stem nuclei regulating antinociception and vascular control.
Perfusion‐weighted imaging defects during spontaneous migrainous aura
TLDR
Functional magnetic resonance imaging can be a useful noninvasive tool to study hemodynamic changes during spontaneous attacks of migraine with aura.
Regional Cerebral Blood Flow and Oxygen Metabolism during Migraine with and Without Aura
TLDR
The data suggest that cerebral ischemia was not the primary cause of the attacks in these cases, and a region corresponding to the primary visual cortex with significant reductions in rCBF and rCMRO2 is revealed.
Focal hyperemia followed by spreading oligemia and impaired activation of rcbf in classic migraine
TLDR
The results indicate that the vasospastic model of the migraine attack is too simplistic and the normal rCBF increase during cortical activity was impaired in 6 patients.
Global cerebral blood flow, blood volume, and oxygen metabolism in patients with migraine headache
TLDR
In patients experiencing migraine without aura, CBF and CBV are reduced during the headache phase, and Cerebral oxygen metabolism and oxygen extraction are not significantly affected.
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