Tissue sparing and functional recovery following experimental traumatic brain injury is provided by treatment with an anti-myelin-associated glycoprotein antibody.

@article{Thompson2006TissueSA,
  title={Tissue sparing and functional recovery following experimental traumatic brain injury is provided by treatment with an anti-myelin-associated glycoprotein antibody.},
  author={Hilaire J Thompson and Niklas Marklund and D Lebold and Diego M. Morales and Carrie A Keck and Mary Vinson and Nicolas C. Royo and Robert I. Grundy and T K Mcintosh},
  journal={The European journal of neuroscience},
  year={2006},
  volume={24 11},
  pages={3063-72}
}
Axonal injury is a hallmark of traumatic brain injury (TBI) and is associated with a poor clinical outcome. Following central nervous system injury, axons regenerate poorly, in part due to the presence of molecules associated with myelin that inhibit axonal outgrowth, including myelin-associated glycoprotein (MAG). The involvement of MAG in neurobehavioral deficits and tissue loss following experimental TBI remains unexplored and was evaluated in the current study using an MAG-specific… CONTINUE READING

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