Tissue Compartment-Specific Estrogen Receptor-α Participation in the Mouse Uterine Epithelial Secretory Response* * Presented in part at the 30th Annual Meeting of the Society for the Study of Reproduction, Portland, Oregon, August 1997. This work was supported by NIH Grants AG-15500 (to P.S.C.), ES-08272 (to D.B.L.), and CA-05388 and AG-13784 (to G.R.C.).

@article{Buchanan1999TissueCE,
  title={Tissue Compartment-Specific Estrogen Receptor-α Participation in the Mouse Uterine Epithelial Secretory Response* * Presented in part at the 30th Annual Meeting of the Society for the Study of Reproduction, Portland, Oregon, August 1997. This work was supported by NIH Grants AG-15500 (to P.S.C.), ES-08272 (to D.B.L.), and CA-05388 and AG-13784 (to G.R.C.).},
  author={David L Buchanan and Tommy Setiawan and Dennis B. Lubahn and J. Andrew Taylor and Takeshi Kurita and Gerald R. Cunha and Paul S. Cooke},
  journal={Endocrinology},
  year={1999},
  volume={140 1},
  pages={484-491}
}
17b-Estradiol (E2) acts through the estrogen receptor (ER) to regulate uterine epithelial cell growth, proliferation, differentiation, and secretory protein production. We have previously shown that E2induced uterine epithelial proliferation is mediated indirectly by ERa-positive stroma; epithelial ERa is neither necessary nor sufficient for E2-induced… CONTINUE READING