The whole truth and nothing but the truth? The research that Philip Morris did not want you to see

  title={The whole truth and nothing but the truth? The research that Philip Morris did not want you to see},
  author={Pascal A. Diethelm and J C Rielle and Martin Mckee},
  journal={The Lancet},

The Tobacco Industry: The Pioneer of Fake News

  • M. Mckee
  • Political Science, Medicine
    Journal of public health research
  • 2017
The tobacco industry poured vast resources into lobbying against smoking bans, employing the classic tactics of denialism to undermine the scientific basis for the bans, and the impact was far greater than almost anyone had anticipated.

A failure of leadership? Why Northern Ireland must introduce a total ban on workplace smoking.

  • M. Mckee
  • Political Science
    The Ulster medical journal
  • 2005
There is, however, a glimmer of hope, as the Minister has stated that there will be a further period of reflection that could lead to a total ban on smoking in public places, given the wealth of experience of the benefits of such bans.

Secret ties to industry and conflicting interests in cancer research.

The shortfalls cited in this article illustrate the need for improved transparency, regulations that will help curb abuses as well as instruments for control and enforcement against abuses.

“A Good Personal Scientific Relationship”: Philip Morris Scientists and the Chulabhorn Research Institute, Bangkok

Previously confidential tobacco industry documents that were made publicly available following litigation in the United States suggest that more rigorous safeguards are required to ensure that such use advances public health goals rather than the objectives of transnational corporations.

Denialism: what is it and how should scientists respond?

The rejection of scientific evidence is also apparent in the popularity of creationism, with an estimated 45% of Americans in 2004 believing that God created man in his present form within the past 10 000 years.

Destroyed documents: uncovering the science that Imperial Tobacco Canada sought to conceal

The scientific evidence contained in the documents destroyed by Imperial Tobacco demonstrates that British American Tobacco had collected evidence that cigarette smoke was carcinogenic and addictive.

Why the European Journal of Public Health will no longer publish tobacco industry-supported research.

The European Journal of Public Health will no longer consider for publication any study that is partly or wholly funded by the tobacco industry. In doing so, it falls into line with the long-standing

Electronic cigarettes: peering through the smokescreen

  • M. Mckee
  • Political Science
    Postgraduate Medical Journal
  • 2014
Health professionals and their patients have every reason to feel confused. One week the WHO issues a position paper calling for strict regulations on electronic nicotine delivery systems (ENDS),

Brussels Declaration: a vehicle for the advancement of tobacco and alcohol industry interests at the science/policy interface?

The Brussels Declaration offers potential to serve as a vehicle for advancing the vested interests of corporate sectors in public policymaking and appears to have been regarded in this way by a range of organisations related to the alcohol industry.

A Longitudinal Assessment of Corrective Advertising Mandated in United States v. Philip Morris USA, Inc.

Results reveal that the corrective ad campaign has not been successful in affecting smokers’ or non-smokers’ antismoking beliefs, and differences are found between smokers�’ and non-Smoking beliefs about the adverse health effects of smoking, effects of secondhand smoke, and tobacco company deceptiveness.



Tobacco industry efforts at discrediting scientific knowledge of environmental tobacco smoke: a review of internal industry documents

Tobacco industry internal documents illustrate a deliberate strategy to use scientific consultants to discredit the science on ETS.

Industry-funded research and conflict of interest: an analysis of research sponsored by the tobacco industry through the Center for Indoor Air Research.

CIAR's stated mission of funding high-quality, objective research has been compromised by conflict of interest, and at least some of CIAR's projects are being used to promote the tobacco industry's agenda.

Lung cancer risk and mutagenicity of tea.

Evaluation of the potential effects of ingredients added to cigarettes. Part 2: chemical composition of mainstream smoke.

Environmental tobacco smoke and tobacco related mortality in a prospective study of Californians, 1960-98

The results do not support a causal relation between environmental Tobacco smoke and tobacco related mortality, although they do not rule out a small effect, and the association between exposure to environmental tobacco smoke and coronary heart disease and lung cancer may be considerably weaker than generally believed.

How the tobacco industry responded to an influential study of the health effects of secondhand smoke

This article documents the tobacco industry's attempts to refute this study by producing a credible alternative study.

Evaluation of the potential effects of ingredients added to cigarettes. Part 3: in vitro genotoxicity and cytotoxicity.

Nicotine and cotinine accumulation in pigmented and unpigmented rat hair.

Hair pigmentation had a major influence on systemic uptake in hair and a minor influence on external uptake inhair, and nicotine and cotinine concentrations in hair were significantly reduced in parallel to the reduced plasma concentrations, showing the influence of metabolism.

Cotinine determination by immunoassays may be influenced by other nicotine metabolites

Polyclonal rabbit anticotinine antiserum, which can be used for biomonitoring nicotine uptake by the determination of cotinine in body fluids, was checked by a competitive ELISA for its

The activity of NF-kappaB in Swiss 3T3 cells exposed to aqueous extracts of cigarette smoke is dependent on thioredoxin.

  • S. GebelT. Müller
  • Biology
    Toxicological sciences : an official journal of the Society of Toxicology
  • 2001
The results suggest that the activity of NF-kappaB in CS-treated cells is subject mainly to a redox-controlled mechanism dependent on the availability of reduced Trx rather than being controlled by its normal regulator, IkappaB-alpha.