The ultrarapid and the transient outward K(+) current in human atrial fibrillation. Their possible role in postoperative atrial fibrillation.

@article{Brandt2000TheUA,
  title={The ultrarapid and the transient outward K(+) current in human atrial fibrillation. Their possible role in postoperative atrial fibrillation.},
  author={Mathias Christoph Brandt and Lars Priebe and Thomas B{\"o}hle and Michael J. Suedkamp and Dirk J. Beuckelmann},
  journal={Journal of molecular and cellular cardiology},
  year={2000},
  volume={32 10},
  pages={
          1885-96
        }
}
Atrial fibrillation (AF) causes distinct changes in atrial conduction, characterized as electrical remodeling. Experimental data on the possible significance of alterations of specific K(+)outward currents in this process are still limited in human AF. The ultra-rapid delayed rectifier current (I(Kur)) has not been studied in AF with respect to its sensitivity to 4-Aminopyridine (4-AP). To clarify the role of (1) the 4-AP sensitive I(Kur)current, compared to recordings without using 4-AP (I(Kur… CONTINUE READING

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