The two mechanisms involved in the control of urinary kallikrein excretion.

Abstract

The relation between sodium and kallikrein excretion is biphasic. Increased kallikrein excretion is produced by increased arterial pressure or arterial infusion of vasodilators. It is antagonised by prostaglandin synthesis inhibition or by arterial noradrenaline infusion. Angiotensin in hypertensive doses increases kallikrein excretion and this effect is… (More)

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