The tripeptide phenylalanine-(d) glutamate-(d) glycine modulates leukocyte infiltration and oxidative damage in rat injured spinal cord

@article{Bao2006TheTP,
  title={The tripeptide phenylalanine-(d) glutamate-(d) glycine modulates leukocyte infiltration and oxidative damage in rat injured spinal cord},
  author={Feng Bao and Sunil M. John and Y. Chen and Ronald D. Mathison and Lynne C. Weaver},
  journal={Neuroscience},
  year={2006},
  volume={140},
  pages={1011-1022}
}

Effects of a novel tripeptide on neurological outcomes after spinal cord injury

The anti-inflammatory and anti-oxidative actions of feG treatment correlate with improved neurological outcomes after spinal cord injury.

The tripeptide feG inhibits leukocyte adhesion

When administered in vivo, feG prevents inflammation-induced reductions in cell adhesion, as well as restoring its inhibitory effect in vitro, and appears to involve actions on αMβ2 integrin, with a possible interaction with the low affinity FcγRIII receptor (CD16).

Preclinical efficacy of Dexmedetomidine on spinal cord injury provoked oxidative renal damage

Investigation of the potential of α2-adrenoreceptor agonist Dexmedetomidine in ameliorating SCI provoked oxidative renal assault finds that Dex may be a beneficial clinical agent to combat post-SCI renal complications.

Beneficial effects of quercetin on rat urinary bladder after spinal cord injury.

Efficacy of coenzyme Q10 in mitigating spinal cord injury-induced osteoporosis.

Treatment with CoQ10 prevented SCI‑induced bone loss by rescuing the decreased levels of bone mineral density and bone mineral content observed in the SCI rats, and reduced bone malondialdehyde levels with a concomitant increase in superoxide dismutase levels, thus alleviating SCI‐induced oxidative injury.

Ghrelin alleviates s pinal Cord injury in Rats Via its anti-inflammatory Effects Ghrelin Anti-inflamatuar Etkileri Yoluyla Siçanlarda Spinal Kord Hasarini Azaltir

The present study suggests that ghrelin could reduce SCI-induced oxidative stress and exert anti-inflammatory effects in the spinal cord following trauma.

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Early anti‐inflammatory treatment reduces lipid peroxidation and protein nitration after spinal cord injury in rats

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