The shift of the reversal potential of glutamate-activated chloride current in molluscan neurons caused by 8-Br-cAMP

Long-term potentiation of glutamatergic synapses is the basis for learning and memory in animals of different species [1–9]. Cyclic adenosine monophosphate (cAMP) plays an important role in the mechanisms of this potentiation. It was shown that an increase in cAMP level in the postsynaptic neuron could cause the potentiation of glutamate synapses in the rat… CONTINUE READING