The interferon signaling network and transcription factor C/EBP-beta.

@article{Li2007TheIS,
  title={The interferon signaling network and transcription factor C/EBP-beta.},
  author={Hui Li and Padmaja Gade and Weihua Xiao and Dhananjaya V R Kalvakolanu},
  journal={Cellular & molecular immunology},
  year={2007},
  volume={4 6},
  pages={
          407-18
        }
}
Cytokines like interferons (IFNs) play a central role in regulating innate and specific immunities against the pathogens and neoplastic cells. A number of signaling pathways are induced in response to IFN in various cells. One classic mechanism employed by IFNs is the JAK-STAT signaling pathway for inducing cellular responses. Here we describe the non-STAT pathways that participate in IFN-induced responses. In particular, we will focus on the role played by transcription factor C/EBP-beta in… CONTINUE READING

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References

Publications referenced by this paper.
SHOWING 1-10 OF 160 REFERENCES

Mechanisms of type-I- and type-II-interferon-mediated signalling

  • Nature Reviews Immunology
  • 2005
VIEW 5 EXCERPTS
HIGHLY INFLUENTIAL

Rap1 signalling: adhering to new models

  • Nature Reviews Molecular Cell Biology
  • 2001
VIEW 5 EXCERPTS
HIGHLY INFLUENTIAL

MEKK1 binds raf-1 and the ERK2 cascade components.

  • The Journal of biological chemistry
  • 2000
VIEW 14 EXCERPTS
HIGHLY INFLUENTIAL

MEKK1 phosphorylates MEK1 and MEK2 but does not cause activation of mitogen-activated protein kinase.

  • Proceedings of the National Academy of Sciences of the United States of America
  • 1995
VIEW 10 EXCERPTS
HIGHLY INFLUENTIAL

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