The effect of Mycoplasma arthritidis infection on the kinetics of colloidal carbon clearance in mice.

@article{Kaklamani1993TheEO,
  title={The effect of Mycoplasma arthritidis infection on the kinetics of colloidal carbon clearance in mice.},
  author={Evangelia Kaklamani and D. Karalis and Y Koumandaki and P. Kaklamanis and E K Katsouyanni and Revekka Tzanetea and C. Caroline Blackwell and L. Sparos and Donald Mackay Weir and Dimitrios Trichopoulos},
  journal={FEMS immunology and medical microbiology},
  year={1993},
  volume={6 4},
  pages={
          299-305
        }
}
The activity of phagocytes from A/J mice was estimated by the carbon clearance test following injection of Mycoplasma arthritidis. Phagocytic activity was significantly depressed 12 h post-infection (P = 0.001) and returned to normal values at 24 h. For animals examined 2 and 7 days post-infection, the overall phagocytic activity increased significantly (P < 10(-4). Phagocytic activity gradually decreased and returned to that of the control group by the end of the fourth week. The relative… 
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TLDR
The phagocytic activity of mononuclear phagocytes of A/J mice and Wistar rats was estimated by the carbon clearance test following injection of Mycoplasma arthritidis and there was not, however, an increase in the relative weight of liver and spleen as observed for the mice.
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TLDR
The clinical course and histopathological characteristics of the chronic phase of mouse arthritis induced by M. arthritidis closely resembled those of rheumatoid arthritis of man.
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TLDR
It was concluded that immunosuppression might be the result of the effect of M. arthritidis on a particular population of cells.
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TLDR
It is suggested that RU-41740 protects the mice by stimulating macrophages, and this immunostimulant might prove useful in the treatment of mycoplasma diseases, especially in the immunocompromised host.
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TLDR
It is hypothesize that MAM may play a role in M. arthritidis-mediated disease by both its inflammatory and immunosuppressive properties as well as major histocompatibility complex restrictions.
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TLDR
It is demonstrated that MAS induces interleukin-6 (IL-6) in murine bone-marrow derived macrophage cultures using C3H/HeJ mice, which are known to be LPS-nonresponders, in all studies.
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TLDR
The kinetics of the clearance of colloidal carbon from the blood of rabbits given an intravenous injection of 10 mg of a tubercle bacillary lipid were investigated and provide a statistical model for a “double exponential” assessment of stimulated carbon clearance rates.
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TLDR
The use of inbred and congenic mouse strains established that toxicity and death induced by Mycoplasma arthritidis associates with the haplotype expressed at the murine major histocompatibility complex, whereas those bearing H-2b are much more resistant.
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