The Beta-Amyloid Protein of Alzheimer's Disease: Communication Breakdown by Modifying the Neuronal Cytoskeleton

@inproceedings{Mokhtar2013TheBP,
  title={The Beta-Amyloid Protein of Alzheimer's Disease: Communication Breakdown by Modifying the Neuronal Cytoskeleton},
  author={Sara H. Mokhtar and Maha M. Bakhuraysah and David S Cram and Steven Petratos},
  booktitle={International journal of Alzheimer's disease},
  year={2013}
}
Alzheimer's disease (AD) is one of the most prevalent severe neurological disorders afflicting our aged population. Cognitive decline, a major symptom exhibited by AD patients, is associated with neuritic dystrophy, a degenerative growth state of neurites. The molecular mechanisms governing neuritic dystrophy remain unclear. Mounting evidence indicates that the AD-causative agent, β -amyloid protein (A β ), induces neuritic dystrophy. Indeed, neuritic dystrophy is commonly found decorating A… CONTINUE READING
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