Th2 cytokines in the asthma late-phase response

@article{Holt2007Th2CI,
  title={Th2 cytokines in the asthma late-phase response},
  author={Patrick G. Holt and Peter D. Sly},
  journal={The Lancet},
  year={2007},
  volume={370},
  pages={1396-1398}
}

A Review of the Pharmacological Properties of Psoralen

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TH9 cell differentiation, transcriptional control and function in inflammation, autoimmune diseases and cancer

It is now clear that TH9 cells play critical roles in immune-mediated diseases, including allergic airway, autoimmune and inflammatory bowel diseases, and cancer, and the promotion or suppression of TH9 cell differentiation, transcriptional control and function may provide novel treatments for clinical inflammation, autoimmune diseases and tumors.

Sesamin attenuates allergic airway inflammation through the suppression of nuclear factor-kappa B activation.

It is demonstrated that sesamin decreases the activation of NF-κB in order to attenuate allergic airway inflammation in a murine model of asthma, possibly via the regulation of phosphorylation of p38 MAPK.

Diagnostic Value of Serum Baseline Tryptase Levels in Childhood Asthma and Its Correlation with Disease Severity

Whether serum baseline tryptase (sBT) levels might be a useful marker not only for the accurate diagnosis of childhood asthma, but also for the prediction of disease severity is explored.

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The results suggested that G-Rh2 attenuates allergic airway inflammation by regulating NF-κB activation and p38 MAPK phosphorylation induced by OVA inhalation, which supported the potential use of G- Rh2 to prevent and/or treat asthma and other airway inflammatory disorders.

The polymeric mucin Muc5ac is required for allergic airway hyperreactivity

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