Systemic inflammatory response following acute myocardial infarction

Abstract

Acute cardiomyocyte necrosis in the infarcted heart generates damage-associated molecular patterns, activating complement and toll-like receptor/interleukin-1 signaling, and triggering an intense inflammatory response. Inflammasomes also recognize danger signals and mediate sterile inflammatory response following acute myocardial infarction (AMI… (More)
DOI: 10.11909/j.issn.1671-5411.2015.03.020

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