Supra- and Infrasylvian Conduction Aphasia

  title={Supra- and Infrasylvian Conduction Aphasia},
  author={Hubertus Axer and A.G.v. Keyserlingk and Georg Berks and Diedrich Graf v. Keyserlingk},
  journal={Brain and Language},
Fifteen cases of conduction aphasia which were tested with the Aachen Aphasia Test (AAT), are presented. The CT lesion data were transformed to a standard 3D-reference brain referring to the ACPC line. According to the lesion profiles a group of 6 patients had pure suprasylvian lesions, a group of 4 patients had pure infrasylvian lesions, and a group of 5 patients had lesions in both supra- and infrasylvian regions. Suprasylvian conduction aphasics are superior to infrasylvian conduction… 
Acute conduction aphasia: An analysis of 20 cases
The role of the arcuate fasciculus in conduction aphasia.
Clinical and neurophysiological findings may suggest that the AF is not required for repetition although could have a subsidiary role in it.
A Review of Conduction Aphasia
  • A. Ardila
  • Psychology
    Current neurology and neuroscience reports
  • 2010
It is concluded that conduction aphasia remains a controversial topic not only from the theoretic point of view, but also from the understanding of its neurologic foundations.
Cortical and fibre tract interrelations in conduction aphasia
First indications for an involvement of the left hemisphere in both short- and long-term integration of auditory information derived by examining patients with (conduction) aphasia are presented.
Conduction aphasia as a function of the dominant posterior perisylvian cortex. Report of two cases.
The authors report two cases that demonstrate that conduction aphasia is cortically mediated and can be inadequately assessed if not specifically evaluated during brain mapping, and a localization of language repetition to the posterior perisylvian cortex.


Conduction aphasia. A clinicopathological study.
Three patients with conduction aphasia are described; in addition to the distinctive language disorder, two of them had severe ideomotor apraxia while the other was entirely free ofApraxia, and a review of the literature would propose the following.
Atypical conduction aphasia. A disconnection syndrome.
Three new cases in which the lesion does not lie in the arcuate fasciculus are reported, offering evidence that disconnection of the circuit linking language comprehension to motor speech output, not damage to a specific cortical region, underlies the syndrome of conduction aphasia.
To what extent can aphasic syndromes be localized?
A retrospective study on 221 aphasic patients with one contiguous vascular lesion in the territory of the middle cerebral artery found no unequivocal association between type of aphasia and localization of lesion, and participation of basal ganglia and in particular of the head of the caudate nucleus in lesions producing aphasIA was not confirmed.
'Inner speech' in conduction aphasia.
This work tested the hypothesis that conduction aphasia is a disturbance of "inner speech" in patients with similar speech disturbances and found that this hypothesis may have been correct for only a subgroup of conductionAphasics.
Conduction aphasia and arcuate fasciculus
Three patients are presented who developed conduction aphasia after a small infarction almost exclusively confined to the arcuate fasciculus, which shows a rapid amelioration within a week after the stroke.
Correlations of subcortical CT lesion sites and aphasia profiles.
The aphasia profiles of 19 cases with subcortical infarction or haemorrhage showed several components of the aphasic syndromes, especially sentence length and grammatical form, ease of speech initiation, articulation, voice volume, and auditory comprehension were individually isolated for correlation with CT lesion site.
Cortical and subcortical aphasias compared
Although some individuals had relative preservation of repetition, it was not confirmed an overall difference between patients with cortical and subcortical lesions in their ability to repeat, and Lesion volume did not significantly correlate with aphasia severity.
Deep left parietal lobe syndrome: conduction aphasia and other neurobehavioural disorders due to a small subcortical lesion.
It is concluded that this tetrad constitutes a specific syndrome which may be easily recognised and ascribed to a single lesion in the deep white matter of the left parietal lobe.