• Corpus ID: 4609485

Stroke caused by varicella zoster virus

@inproceedings{Nagel2014StrokeCB,
  title={Stroke caused by varicella zoster virus},
  author={Maria A. Nagel and Donald H. Gilden and Martin S. Hirsch},
  year={2014}
}
This topic will review the pathogenesis, epidemiology, risk factors for, clinical features, diagnosis, and treatment of VZV vasculopathy. The major clinical manifestations and complications of chickenpox and herpes zoster are discussed separately. (See "Epidemiology of varicella-zoster virus infection: Chickenpox" and "Treatment of varicella-zoster virus infection: Chickenpox" and "Vaccination for the prevention of varicella-zoster virus infection: Chickenpox" and "Clinical manifestations of… 

References

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TLDR
Varicella-zoster virus infects a wide variety of cell types in the central and peripheral nervous system, explaining the diversity of clinical disorders associated with the virus.

Pathogenesis of varicella-zoster angiitis in the CNS.

A 20-year-old man with Hodgkin's disease experienced ophthalmic zoster with dissemination and CNS involvement. At autopsy, he was found to have granulomatous angiitis involving the basilar artery,

Thrombotic cerebral vasculopathy associated with herpes zoster

TLDR
Evidence is provided that the vasculopathy following herpes zoster may result from direct VZV infection of the artery and the in situ thrombosis can develop within the infected vessels in the absence of clear inflammatory vasculitis.

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We report a case of varicella-zoster vasculopathy that occurred in a 42-year-old renal transplant recipient with concurrent vertebral artery aneurysm and dissection. The patient was successfully

Varicella zoster virus, a cause of waxing and waning vasculitis

TLDR
A search for VZV should be conducted in cases of vasculitis when both the central and peripheral nervous systems are involved, when focal narrowing is present in large arteries, when brain imaging reveals infarction in gray and white matter, both deep and superficial, and when white matter is disproportionally involved.

The vasculopathy of varicella‐zoster virus encephalitis

Varicella‐zoster virus (VZV) encephalitis has become more prevalent in the era of acquired immunodeficiency syndrome and other immunosuppressive diseases and poses diagnostic and therapeutic

Unusual presentation of central nervous system manifestations of Varicella zoster virus vasculopathy in renal transplant recipients

TLDR
This is the first description of cerebral VZV vasculopathy in solid organ transplant recipients with severe but reversible neurological manifestations related to Varicella zoster virus (VZV) cerebral Vasculopathy.

Varicella-zoster virus infection of the central nervous system in the acquired immune deficiency syndrome.

TLDR
It is suggested that VZV infection of the CNS occurs more frequently in AIDS than previously suspected and suggests that it must be considered as a diagnosis in cases of encephalitis, ventriculitis, focal myelitis, acute myeloradiculitis and cerebral infarcts in these patients.

The varicella zoster virus vasculopathies

TLDR
Rash or CSF pleocytosis is not required to diagnose varicella zoster virus (VZV) vasculopathy, whereas MRI/CT abnormalities are seen in almost all patients, and determination of optimal antiviral treatment and benefit of concurrent steroid therapy awaits studies with larger case numbers.