Sprint training normalizes Ca(2+) transients and SR function in postinfarction rat myocytes.

@article{Zhang2000SprintTN,
  title={Sprint training normalizes Ca(2+) transients and SR function in postinfarction rat myocytes.},
  author={Lianqin Zhang and X Q Zhang and Yon Chiet Ng and L. I. Rothblum and Timothy I Musch and Russell Moore and Joseph Y Cheung},
  journal={Journal of applied physiology},
  year={2000},
  volume={89 1},
  pages={38-46}
}
Previous studies have shown that myocytes isolated from sedentary (Sed) rat hearts 3 wk after myocardial infarction (MI) undergo hypertrophy, exhibit altered intracellular Ca(2+) concentration ([Ca(2+)](i)) dynamics and abnormal contraction, and impaired sarcoplasmic reticulum (SR) function manifested as prolonged half-time of [Ca(2+)](i) decline. Because… CONTINUE READING