Some properties of mouse platelets.

@article{Rosenblum1983SomePO,
  title={Some properties of mouse platelets.},
  author={W I Rosenblum and Guy H. Nelson and Carolyn S. Cockrell and Earl F. Ellis},
  journal={Thrombosis research},
  year={1983},
  volume={30 4},
  pages={
          347-55
        }
}
Comparative Physiology of Platelets from Different Species
The mammalian blood platelet contributes to hemostasis as a result of its ability to adhere to exposed subendothelial collagen and to respond to stimuli, including, thromobin and adenosine
Characterization of the aggregation responses of camel platelets.
TLDR
Whether camel platelets can be activated by high doses or combinations of aggregation agonists, and to characterize the receptor that mediates the aggregation response to adenosine diphosphate (ADP), the most potent agonist for camel platelet aggregation known so far.
Angiotensin delays platelet aggregation after injury of cerebral arterioles.
TLDR
The data represent the first report of an antiaggregating effect of angiotensin II in vivo in an intact microvascular bed and are consonant with the literature describing increased prostacyclin levels following angiotENSin II infusion.
Defective platelet activation in Gαq-deficient mice
TLDR
It is shown that platelets from mice deficient in the α-subunit of the heterotrimeric guanine-nucleotide-binding protein Gq are unresponsive to a variety of physiological platelet activators, and that Gαq is essential for the signalling processes used by different platelet Activators and that it cannot be replaced by Gαi or the βγ subunits of the homophobic G proteins.
Effects of estradiol on platelet aggregation in cerebral microvessels of mice.
TLDR
The data are discussed in light of the literature suggesting enhancement of ischemic vascular disease, including strokes, in patients receiving estrogens, and especially high doses of estrogens.
Platelet aggregation and platelet function analyzer 100 (PFA-100) closure time in camels—a comparative study with humans
TLDR
The lack of platelet responses to certain aggregating agonists, their enhancement with human plasma and serum, as well as the prolongation of the PFA-100 closure times, add other unique characteristics to the biology of this interesting creature.
The P2Y1 receptor is essential for ADP-induced shape change and aggregation in mouse platelets.
TLDR
The findings clearly demonstrate the existence of two distinct ADP receptors, the P2Y1 and P2T(AC), in mouse platelets with similar function as in human platelets.
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References

SHOWING 1-10 OF 16 REFERENCES
Aggregation of platelets of normal and Bordetella pertussis vaccinated mice by ATP, ADP, AND sodium fluoride.
TLDR
The results of the present studies have shown that platelets obtained from B. pertussis vaccinated mice were more sensitive to the aggregative action of ATP, ADP, and sodium fluoride than were their normal counterparts, which suggested a role of adenyl cyclase and adrenergic receptors in the mouse platelet aggregation.
THE INFLUENCE OF CITRATE AND HEPARIN ON THE ADHESIVENESS OF RAT PLATELETS AND HUMAN PLATELETS MEASURED IN VITRO.
  • A. Nordøy, A. Odegaard
  • Biology, Medicine
    Scandinavian journal of clinical and laboratory investigation
  • 1963
1. The influence of heparin and citrate on the platelet adhesiveness to glass of normal rat platelets and normal human platelets in whole blood has been examined in vitro.2. When citrate in a final
Interrelations of platelet aggregation and secretion.
TLDR
The mechanism of stimulus-response coupling in human platelets was investigated with a new instrument that simultaneously monitors aggregation and secretion in the same sample of plateletrich plasma, finding little evidence for second wave aggregation being a consequence of secretion as usually described.
Heparin inhibits PGE1-sensitive adenylate cyclase and antagonizes PGE1 antiaggregating effect in human platelets.
TLDR
PGE1-sensitive adenylate cyclase in human platelet lysates in inhibited (50%) by heparin (10 microgram/ml), which effectively antagonizes the antiaggregating effect of PGE1 on ADP-induced platelet aggregation.
Proaggregatory Effect of Fasting on Platelet Aggregation in the Microcirculation of Mice with Streptozotocin Diabetes
TLDR
The capacity to enhance aggregation at will by fasting streptozotocin diabetics may provide a means by which some of the factors controlling aggregation In this model of diabetes can be identified, and this in turn may provide insights into the reasons for the variable occurrence of enhanced aggregation in other species or In other types of diabetes.
Arachidonate‐induced cerebrovascular occlusion in the rat
TLDR
A neurologic syndrome, including ipsilateral blindness and contralateral sensorimotor deficits, resulted from occlusion of the microvasculature by platelet thrombi, and the drug offered little protection against arachidonate-induced stroke.
Arachidonic acid metabolites and the interactions between platelets and blood-vessel walls.
PROSTAGLANDINS are potent vasoactive agents with a wide variety of other actions that depend on the species and organ tested and the prostaglandin used. They are synthesized from 20-carbon polyunsa...
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