Sobre a posição do ureter no hilo renal de bovinos da raça Hereford

  title={Sobre a posiç{\~a}o do ureter no hilo renal de bovinos da raça Hereford},
  author={Althen Teixeira Filho and Ant{\^o}nio Fernandes Filho and Jos{\'e} Peduti Neto},
  journal={Brazilian Journal of Veterinary Research and Animal Science},
There was studied the position of the emergence of the ureter in the hilus in 30 pairs of kidneys of Hereford cattle. For that, the hilus was divided in quadrants so named: craniolateralis, craniomedialis, caudolateralis and caudomedialis. The results obtained show different sites of emergence, especially in the left kidney. 
13 Citations
T lymphocyte immunophenotypes in the cerebrospinal fluid of dogs with visceral leishmaniasis.
In insight into the immunophenotypes of T lymphocytes in the CSF during canine visceral leishmaniasis, the results provide more explanation to the inflammation observed in the brain of dogs with VL, which the DN T cells contribute to the origin and progression of the neurological disease.
T and B lymphocytes in the brains of dogs with concomitant seropositivity to three pathogenic protozoans: Leishmania chagasi, Toxoplasma gondii and Neospora caninum
It is demonstrated that the peripheral infection by L. chagasi per se can promote the influx of lymphocytes within the nervous milieu as occurs during Toxoplasma and Neospora infections, and the concomitant seropositivity against these pathogens does not exacerbate the inflammatory brain lesions.
High levels of serum matrix metalloproteinases in dogs with natural visceral leishmaniosis: a preliminary report.
The findings suggest that the multi-systemic inflammatory lesions observed in visceral leishmaniosis are associated with an increase in serum MMPs, especially MMP-9.
First detection of Leishmania infantum DNA within the brain of naturally infected dogs.
For the first time, the presence of parasite DNA was detected and quantified in the brains of naturally infected dogs, in all evaluated regions, and contributes to a new perspective of the pathogenesis of visceral leishmaniasis.
Levels of matrix metalloproteinase‐2 and metalloproteinase‐9 in the cerebrospinal fluid of dogs with visceral leishmaniasis
The presence and persistence of MMPs in CSF supports the hypothesis of an inflammatory state within CNS of dogs with VL, and suggests that MMP‐9 might play a role in disruption of blood–brain barrier and/or blood–CSF barrier.
Glial reactivity in dogs with visceral leishmaniasis: correlation with T lymphocyte infiltration and with cerebrospinal fluid anti-Leishmania antibody titres
Results indicate a pro-inflammatory state in the brains of dogs naturally infected with L. chagasi and strongly suggest that microglia and astrocytes are involved in the pathogenesis of the neurological disorders of visceral leishmaniasis in dogs.
Canine cerebral leishmaniasis: potential role of matrix metalloproteinase-2 in the development of neurological disease.
Although the detected MMP activity in the CSF might merely be related to CNS inflammation, these enzymes may also play a collaborative role in the disease progression, and once activated, they may promote cerebral barrier breakdown.
Leishmania infection and neuroinflammation: Specific chemokine profile and absence of parasites in the brain of naturally-infected dogs
Not necessarily the parasite itself, but rather its DNA seems to act as a trigger to promote brain inflammation during visceral leishmaniasis.
Toll-like receptors and cytokines in the brain and in spleen of dogs with visceral leishmaniosis.
This study provides the first insight into the Toll-like receptors profile in the brain and the spleen during canine VL and provides support to confirm the involvement of sensors of the innate immune system sensors against L. infantum parasites.
Pro-inflammatory cytokines predominate in the brains of dogs with visceral leishmaniasis: a natural model of neuroinflammation during systemic parasitic infection.
The data indicate the presence of a pro-inflammatory status in the nervous milieu of dogs with visceral leishmaniasis especially because IL-1β and TNF-α are considered key factors for the initiation, maintenance and persistence of inflammation.