Smoking, COPD, and 3-Nitrotyrosine Levels of Plasma Proteins

@inproceedings{Jin2011SmokingCA,
  title={Smoking, COPD, and 3-Nitrotyrosine Levels of Plasma Proteins},
  author={Hongjun Jin and Bobbie-Jo M. Webb-Robertson and Elena S. Peterson and Ruimin M Tan and Diana J. Bigelow and Mary Beth Scholand and John R. Hoidal and Joel G. Pounds and Richard C. Zangar},
  booktitle={Environmental health perspectives},
  year={2011}
}
BACKGROUND Nitric oxide is a physiological regulator of endothelial function and hemodynamics. Oxidized products of nitric oxide can form nitrotyrosine, which is a marker of nitrative stress. Cigarette smoking decreases exhaled nitric oxide, and the underlying mechanism may be important in the cardiovascular toxicity of smoking. Even so, it is unclear if this effect results from decreased nitric oxide production or increased oxidative degradation of nitric oxide to reactive nitrating species… CONTINUE READING

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