Selenium toxicity: cause and effects in aquatic birds.

@article{Spallholz2002SeleniumTC,
  title={Selenium toxicity: cause and effects in aquatic birds.},
  author={Julian E. Spallholz and David J. Hoffman},
  journal={Aquatic toxicology},
  year={2002},
  volume={57 1-2},
  pages={
          27-37
        },
  url={https://api.semanticscholar.org/CorpusID:28251689}
}

Prooxidant mechanisms of selenium toxicity - A review

Exact mechanism of selenium toxicity remains unclear but there are many data about its prooxidant effect particularly in the form ofselenite, while selenomethionine and selenocysteine are less toxic.

Distribution and metabolism of selenite and selenomethionine in the Japanese quail.

The highest concentration of Se was detected in the pancreas, followed by down feathers, liver, and kidneys, and SeMet was more efficiently incorporated into the quail than selenite, and selenosugar and trimethylselenonium were the major metabolites in quail excreta.

The Properties, Functions, and Use of Selenium Compounds in Living Organisms

This article gathers information from the literature on the consequences of a deficiency and an excess of selenium in the body, as well as the health-promoting mechanisms of Selenium, including the functions ofSelenoproteins.

Selenium Pollution in the Marine Environment and Marine Bacteria in Selenium Bioremediation

Recent studies suggest the use of microbiological resources to detoxify selenium to be the most simple and economical method, and the conventional physicochemical methods employed in seenium removal, although effective, may prove to be quite expensive.
...

[Selenium methylation and toxicity mechanism of selenocystine].

This review summarized recent studies on the toxicity mechanism of selenocystine in experimental animals and found that accumulation of the hydrogen selenide resulting from inhibition of the selenium methylation metabolism, detoxification metabolic pathway of seenium, is found in animals following repeated administration of a toxic dose of selecystine.

Toxicity and oxidative stress of different forms of organic selenium and dietary protein in mallard ducklings

Se as L at 30 ppm in the diet was the most toxic form, resulting in high mortality and impaired growth in survivors and the greatest increase in ratio of oxidized to reduced hepatic glutathione (GSH).

Selenium in Plants: Uptake, Functions, and Environmental Toxicity

Biogeochemical cycling of Se involves significant volatilization of methylated selenides such as dimethyl selenide to the atmosphere from higher plants as well as freshwater algae, but Se exchange between oceans and the atmosphere appears to proceed as net flux to the oceans.

Effects of mercury and selenium on glutathione metabolism and oxidative stress in mallard ducks

The ability of Se to restore the activities of G‐6‐PDH, GSH peroxidase, and glutathione status involved in antioxidative defense mechanisms may be crucial to biological protection from the toxic effects of methylmercury.

Hepatic glutathione metabolism and lipid peroxidation in response to excess dietary selenomethionine and selenite in mallard ducklings.

Excess dietary Se as selenomethionine has a more pronounced effect on hepatic glutathione metabolism and lipid peroxidation in ducklings than does selenite, which may be related to the pattern of accumulation.

Selenium toxicosis in wild aquatic birds.

It is concluded that selenium present in the agricultural drainage water supplied to the Kesterson ponds accumulated in the food chain of aquatic birds to toxic concentrations and caused the lesion and other changes observed.

Interactive effects of arsenate, selenium, and dietary protein on survival, growth, and physiology in mallard ducklings

The findings suggest the potential for antagonistic effects of Se and As on duckling survival, growth, and physiology with adequate dietary protein but more severe toxicological effects when dietary protein is diminished.
...