Role of lung apolipoprotein A-I in idiopathic pulmonary fibrosis: antiinflammatory and antifibrotic effect on experimental lung injury and fibrosis.

@article{Kim2010RoleOL,
  title={Role of lung apolipoprotein A-I in idiopathic pulmonary fibrosis: antiinflammatory and antifibrotic effect on experimental lung injury and fibrosis.},
  author={Tae Hoon Kim and Yoo Hoon Lee and Kyung Hun Kim and Shin Hwa Lee and Ji Yeon Cha and Eun Kyoung Shin and Seok Ho Jung and An Soo Jang and Sung Woo Park and Soo Taek Uh and Young Hoon Kim and Jai Soung Park and Hwa Gyoun Sin and Wook Youm and Eun suk Koh and Sun Young Cho and Young Ki Paik and Tai Youn Rhim and Choon Sik Park},
  journal={American journal of respiratory and critical care medicine},
  year={2010},
  volume={182 5},
  pages={633-42}
}
RATIONALE Idiopathic pulmonary fibrosis (IPF) is caused by alterations in expression of proteins involved in multiple pathways, including matrix deposition, inflammation, injury, and repair. OBJECTIVES To understand the pathogenic changes in lung protein expression in IPF and to evaluate apolipoprotein (Apo) A-I as a candidate therapeutic molecule. METHODS Two-dimensional electrophoresis was adopted for differential display proteomics. Reverse-transcriptase polymerase chain reaction… CONTINUE READING
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