Role of endogenous nitric oxide in ischemia-reperfusion injury in rat liver.

@article{Kobayashi1995RoleOE,
  title={Role of endogenous nitric oxide in ischemia-reperfusion injury in rat liver.},
  author={Honami Kobayashi and Toshiaki Nonami and Toru Kurokawa and Yoji Takeuchi and Akio Harada and Akimasa Nakao and Hitoshi Takagi},
  journal={The Journal of surgical research},
  year={1995},
  volume={59 6},
  pages={772-9}
}
Evidence has accumulated that oxygen-derived free radicals contribute to the cellular damage induced by ischemia-reperfusion. It has been suggested that nitric oxide (NO) may act as a protective factor in ischemia-reperfusion injury since NO increases blood flow and may scavenge oxyradicals. Nevertheless, controversy exists as to the role of NO. This study was designed to clarify the role of endogenous NO in ischemia-reperfusion-induced liver injury in rats in vivo. Wistar rats weighing 250-300… CONTINUE READING
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