• Corpus ID: 36996869

Review of azotemia in foals.

  title={Review of azotemia in foals.},
  author={Harold C Schott},
Azotemia in neonatal foals ( 7 days of age) may be an indicator of pre-renal failure, acute kidney injury (AKI), obstructive disease, congenital renal disorders, or disruption of the collecting system leading to uroperitoneum. Because clinical signs of renal failure may be similar to those with septicemia or asphyxia (weakness, recumbency, or poor nursing vigor), performing a serum biochemical analysis in compromised neonates is an essential part of the minimum database to detect azotemia… 
Acompanhamento clínico de potro neonato proveniente de gestação com placentite Clinical Monitoring of Neonatal Foal from Pregnancy with Placentitis
It is concluded that the delivery monitoring and the follow-up of the foal allows an early diagnosis of dysmaturity and the determination of absence of sepsis, guiding the therapeutic approach and management measures that can lead to the survival of thefoal.
Oral single dose of allopurinol in thoroughbred foals born from mares with placentitis
Administration of Allopurinol PO in foals born from mares with placentitis did not result in adverse effects and can help in stabilizing serum calcium and glucose levels.
Newborn adaptations and healthcare throughout the first age of the foal
This process includes a detailed evaluation of the mare during pregnancy and foaling, as well as clinical observations of neurological reflexes from the neonatal foal, behavioral and laboratory tests to determine the degree of maturity and viability of the horse.


Uroperitoneum in 32 foals: influence of intravenous fluid therapy, infection, and sepsis.
Intrauterine compromise, presumed hypoxia or ischemia, and sepsis may predispose foals to development of uroperitoneum, while foals referred with suspected UP often had additional problems unrelated to the urinary system.
Spurious hypercreatininemia: 28 neonatal foals (2000-2008).
Creatinine decreased by >50% within the initial 24 hours of standard neonatal therapy and was within the reference interval in all but 1 foal within 72 hours of hospitalization.
Acute kidney injury in children
Improved understanding of the pathophysiology of AKI, early biomarkers ofAKI, and better classification of AKi are needed for the development of successful therapeutic strategies for the treatment of AK I.
Indices of renal function: values in eight normal foals from birth to 56 days.
Blood chemistry relating to renal function was evaluated as well as physical and chemical characteristics of urine and there was some variation with time, generally the urinary activity of gamma-glutamyl transpeptidase and alkaline phosphatase was greater in foals than in adults.
Acute kidney injury post neonatal asphyxia.
The current evidence regarding the epidemiology, investigation, and treatment of AKI in the asphyxiated neonate is discussed, with particular emphasis given to the investigation of renal function in the neonate and to potential biomarkers that may aid the clinician in the diagnosis of renal injury in this population.
Nephrotoxicity as a cause of acute kidney injury in children
Nonsteroidal anti-inflammatory drugs (NSAIDs), antibiotics, amphotericin B, antiviral agents, angiotensin-converting enzyme (ACE) inhibitors, calcineurin inhibitors, radiocontrast media, and cytostatics are the most important drugs to indicate AKI as significant risk factor in children.
Acute renal disease from Leptospira interrogans in three yearlings from the same farm
3 horses with acute renal disease from Leptospira interrogans developed an acute and persistent febrile illness that could have been attributed to a number of infectious diseases.
Hemodialysis for treatment of oxytetracycline-induced acute renal failure in a neonatal foal.
Hemodialysis may be a treatment option in management of acute renal failure in foals that are nonresponsive to conservative medical treatment and the nephrotoxic potential of oxytetracycline should be considered prior to its administration.
Fibrin deposits and organ failure in newborn foals with severe septicemia.
Nonsurviving septic foals have fibrin deposits in their tissues, a finding consistent with capillary microthrombosis and DIC.
Renal clearance, urinary excretion of endogenous substances, and urinary diagnostic indices in healthy neonatal foals.
It was concluded that the use of single sample urine/serum estimates of fractional excretion in the neonatal foal was an appropriate indicator of the renal handling of electrolytes, and when viewed in conjunction with urinalysis and other serum parameters, a valuable aid to evaluating renal function.