Reversibility of promoter induced hepatic focal lesion growth in mice.

@article{Kolaja1996ReversibilityOP,
  title={Reversibility of promoter induced hepatic focal lesion growth in mice.},
  author={K. Kolaja and D. E. Stevenson and E. Walborg and J. Klaunig},
  journal={Carcinogenesis},
  year={1996},
  volume={17 7},
  pages={
          1403-9
        }
}
The effect of cessation of phenobarbital and dieldren treatment on hepatic focal lesion growth in male B6C3F1 mice was investigated. Following induction of lesions by diethylnitrosamine, mice were placed on control NIH-07 diet (control diet) or NIH-07 diet containing either dieldrin (10.0 mg/kg diet) or phenobarbital (500 mg/kg diet). Mice were sacrificed after 30 and 60 days of dietary treatment. Two additional groups of mice were fed either the dieldren- or phenobarbital-containing diet for… Expand
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Short-term fasting and the reversal of the stage of promotion in rat hepatocarcinogenesis: role of cell replication, apoptosis, and gene expression.
Effect of antisense oligomer in controlling c-raf.1 overexpression during diethylnitrosamine-induced hepatocarcinogenesis in rat
Biological Basis of Differential Susceptibility to Hepatocarcinogenesis among Mouse Strains*
  • R. Maronpot
  • Biology, Medicine
  • Journal of toxicologic pathology
  • 2009
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