Retinal dysplasia and degeneration in RARbeta2/RARgamma2 compound mutant mice.

@article{Grondona1996RetinalDA,
  title={Retinal dysplasia and degeneration in RARbeta2/RARgamma2 compound mutant mice.},
  author={Jesus M. Grondona and Philippe Kastner and Anne Gansmuller and Didier D{\'e}cimo and Pierre Chambon and Manuel Mark},
  journal={Development},
  year={1996},
  volume={122 7},
  pages={
          2173-88
        }
}
The eye is the organ whose development is the most frequently altered in response to maternal vitamin A deficiency [VAD; Warkany, J. and Schraffenberger, S. (1946). Archs Ophthalmol. 35, 150-169]. With the exception of prenatal retinal dysplasia, all the ocular abnormalities of the fetal VAD syndrome are recapitulated in mouse mutants lacking either RARalpha and RARbeta2, RARalpha and RARgamma, RARgamma and RARbeta2, or RXRalpha [Lohnes, D., Mark, M., Mendelsohn, C., Dolle, P., Dierich, A… CONTINUE READING

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