Retention and extinction of delay eyeblink conditioning are modulated by central cannabinoids.

@article{Steinmetz2011RetentionAE,
  title={Retention and extinction of delay eyeblink conditioning are modulated by central cannabinoids.},
  author={Adam B Steinmetz and John H. Freeman},
  journal={Learning & memory},
  year={2011},
  volume={18 10},
  pages={634-8}
}
Rats administered the cannabinoid agonist WIN55,212-2 or the antagonist SR141716A exhibit marked deficits during acquisition of delay eyeblink conditioning, as noted by Steinmetz and Freeman in an earlier study. However, the effects of these drugs on retention and extinction of eyeblink conditioning have not been assessed. The present study examined the effects of WIN55,212-2 and SR141716A on retention and extinction of delay eyeblink conditioning in rats. Rats were given acquisition training… CONTINUE READING
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