Renal tubular angiogenic dysregulation in anti-Thy1.1 glomerulonephritis.

Abstract

Peritubular vascular changes and hypoxia after glomerular injury may explain subsequent tubulointerstitial injury and fibrosis. Several studies suggested that the expected tubulointerstitial angiogenic response is actively suppressed in this setting. The mechanism of this aberrant response has not been clearly identified. We used a common model of… (More)
DOI: 10.1152/ajprenal.00214.2010

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