Relevance of hereditary defects in lipid transport proteins for the pathogenesis of cholesterol gallstone disease.

Abstract

In the formation of cholesterol gallstones, cholesterol hypersecretion into bile causing cholesterol supersaturation and crystallization appears to be the primary factor, with disturbed gallbladder and intestinal motility as secondary factors. Although intestinal uptake mechanisms have not yet been fully elucidated, the HDL receptor scavenger receptor B1… (More)

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