Reduced glutathione prevents nitric oxide-induced apoptosis in vascular smooth muscle cells.

Association of the ERK1/2 and p38 kinase pathways with nitric oxide-induced apoptosis and cell cycle arrest in colon cancer cells

Gain and loss of function for glutathione synthesis: impact on advanced atherosclerosis in apolipoprotein E-deficient mice.

Modulation of YY1 and p53 expression by transforming growth factor-β3 in prostate cell lines.

Phosphodiesterase function and endocrine cells: links to human disease and roles in tumor development and treatment.

Nitric oxide stimulates heme oxygenase-1 gene transcription via the Nrf2/ARE complex to promote vascular smooth muscle cell survival.

Nuclear Translocation of Components of the Neuronal Plasma Membrane Oxidoreductase after Oxidative Stress and its Effects on Induction of Apoptosis

SOXF: redox mediators of vascular smooth muscle cell growth.

[Programmed cellular death and atherogenesis: from molecular mechanisms to clinical aspects].

Thalidomide modulates nuclear redox status and preferentially depletes glutathione in rabbit limb versus rat limb.

Cyclophilin A is a secreted growth factor induced by oxidative stress.

Reduced glutathione prevents nitric oxide-induced apoptosis in vascular smooth muscle cells.

Association of the ERK1/2 and p38 kinase pathways with nitric oxide-induced apoptosis and cell cycle arrest in colon cancer cells

Gain and loss of function for glutathione synthesis: impact on advanced atherosclerosis in apolipoprotein E-deficient mice.

Modulation of YY1 and p53 expression by transforming growth factor-β3 in prostate cell lines.

Phosphodiesterase function and endocrine cells: links to human disease and roles in tumor development and treatment.

Nitric oxide stimulates heme oxygenase-1 gene transcription via the Nrf2/ARE complex to promote vascular smooth muscle cell survival.

Nuclear Translocation of Components of the Neuronal Plasma Membrane Oxidoreductase after Oxidative Stress and its Effects on Induction of Apoptosis

SOXF: redox mediators of vascular smooth muscle cell growth.

[Programmed cellular death and atherogenesis: from molecular mechanisms to clinical aspects].

Thalidomide modulates nuclear redox status and preferentially depletes glutathione in rabbit limb versus rat limb.

Cyclophilin A is a secreted growth factor induced by oxidative stress.

Reduced glutathione prevents nitric oxide-induced apoptosis in vascular smooth muscle cells.

Association of the ERK1/2 and p38 kinase pathways with nitric oxide-induced apoptosis and cell cycle arrest in colon cancer cells

Gain and loss of function for glutathione synthesis: impact on advanced atherosclerosis in apolipoprotein E-deficient mice.

Modulation of YY1 and p53 expression by transforming growth factor-β3 in prostate cell lines.

Phosphodiesterase function and endocrine cells: links to human disease and roles in tumor development and treatment.

Nitric oxide stimulates heme oxygenase-1 gene transcription via the Nrf2/ARE complex to promote vascular smooth muscle cell survival.

Nuclear Translocation of Components of the Neuronal Plasma Membrane Oxidoreductase after Oxidative Stress and its Effects on Induction of Apoptosis

SOXF: redox mediators of vascular smooth muscle cell growth.

[Programmed cellular death and atherogenesis: from molecular mechanisms to clinical aspects].

Thalidomide modulates nuclear redox status and preferentially depletes glutathione in rabbit limb versus rat limb.

Cyclophilin A is a secreted growth factor induced by oxidative stress.