Recovery from ketamine-induced amnesia by blockade of GABA-A receptor in the medial prefrontal cortex of mice

@article{Farahmandfar2017RecoveryFK,
  title={Recovery from ketamine-induced amnesia by blockade of GABA-A receptor in the medial prefrontal cortex of mice},
  author={Maryam Farahmandfar and Ardeshir Akbarabadi and Atefeh Bakhtazad and Mohammad Reza Zarrindast},
  journal={Neuroscience},
  year={2017},
  volume={344},
  pages={48-55}
}

Cortical and raphe GABAA, AMPA receptors and glial GLT-1 glutamate transporter contribute to the sustained antidepressant activity of ketamine

TLDR
The results suggest the need to test for the concomitant prescription of ketamine and BZD to see whether its sustained antidepressant activity is maintained in TRD patients, and highlight the role of neuronal-glial adaptation in these effects.

Blockage of NMDA- and GABA(A) Receptors Improves Working Memory Selectivity of Primate Prefrontal Neurons

The ongoing activity of prefrontal neurons after a stimulus has disappeared is considered a neuronal correlate of working memory. It depends on the delicate but poorly understood interplay between

Ketamine abuse potential and use disorder

GluN2A-selective positive allosteric modulator-nalmefene-flumazenil reverses ketamine-fentanyl-dexmedetomidine-induced anesthesia and analgesia in rats

TLDR
The potential application of the PNF combination with PAM combined with nalmefene and flumazenil in reversing the actions of an anesthetic combination is suggested.

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