Reactive oxygen species activate HIV long terminal repeat via post-translational control of NF-kappaB.

@article{Pyo2008ReactiveOS,
  title={Reactive oxygen species activate HIV long terminal repeat via post-translational control of NF-kappaB.},
  author={Chul-Woong Pyo and Young Yang and Na-Kyung Yoo and S Choi},
  journal={Biochemical and biophysical research communications},
  year={2008},
  volume={376 1},
  pages={180-5}
}
Reduction/oxidation disorder is one of the most common ailments in HIV-infected patients, and such patients are frequently left exposed to chronic oxidative stress after the generation of reactive oxygen species (ROS). Although a variety of clinical trials to inhibit HIV infection have been conducted by focusing on oxidative stress, their precise targets and reaction mechanism have remained unclear. In this study, we demonstrate that H2O2 treatment strongly induced HIV long terminal repeat (LTR… CONTINUE READING
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