Reactive oxygen species/oxidative stress contributes to progression of kidney fibrosis following transient ischemic injury in mice.

@article{Kim2009ReactiveOS,
  title={Reactive oxygen species/oxidative stress contributes to progression of kidney fibrosis following transient ischemic injury in mice.},
  author={Jinu Kim and Young Mi Seok and Kyong-Jin Jung and Kwon Moo Park},
  journal={American journal of physiology. Renal physiology},
  year={2009},
  volume={297 2},
  pages={F461-70}
}
Recently, kidney fibrosis following transplantation has become recognized as a main contributor of chronic allograft nephropathy. In transplantation, transient ischemia is an inescapable event. Reactive oxygen species (ROS) play a critical role in ischemia and reperfusion (I/R)-induced acute kidney injury, as well as progression of fibrosis in various diseases such as hypertension, diabetes, and ureteral obstruction. However, a role of ROS/oxidative stress in chronic kidney fibrosis following I… CONTINUE READING

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